[Hemodynamic effects of intranasal nifedipine during clamping of the abdominal aorta]

Ann Fr Anesth Reanim. 1987;6(2):79-82. doi: 10.1016/s0750-7658(87)80107-7.
[Article in French]

Abstract

The haemodynamic changes due to cross-clamping of the abdominal aorta below the renal arteries were studied in ten patients. Anaesthesia was induced with thiopentone and maintained with fentanyl and vecuronium and inhalation of 60% nitrous oxide in oxygen. At the fifth minute, clamping increased mean arterial pressure (Pa) by 11%, systemic vascular resistance (Rsa) by 26% and decreased cardiac output (CO) by 20%. Nifedipine was administered intranasally at this time. Heart rate remained unchanged; mean pulmonary arterial and mean pulmonary wedge pressures were slightly decreased. Pa and Rsa fell to significantly lower levels between the fifth and fifteenth minutes (24 and 43% respectively). Although CO increased by 28%, this was not significant. The administration of intranasal nifedipine during anaesthesia was well tolerated. This study demonstrated that intranasal nifedipine prevented adverse haemodynamic effects of cross-clamping of the aorta below the renal arteries.

Publication types

  • English Abstract

MeSH terms

  • Administration, Intranasal
  • Anesthesia, General
  • Aorta, Abdominal / surgery*
  • Constriction
  • Female
  • Hemodynamics / drug effects*
  • Humans
  • Male
  • Nifedipine / administration & dosage
  • Nifedipine / pharmacology*

Substances

  • Nifedipine