FtsH degrades kinetically stable dimers of cyclopropane fatty acid synthase via an internal degron

Mol Microbiol. 2023 Jan;119(1):101-111. doi: 10.1111/mmi.15009. Epub 2022 Dec 14.

Abstract

Targeted protein degradation plays important roles in stress responses in all cells. In E. coli, the membrane-bound AAA+ FtsH protease degrades cytoplasmic and membrane proteins. Here, we demonstrate that FtsH degrades cyclopropane fatty acid (CFA) synthase, whose synthesis is induced upon nutrient deprivation and entry into stationary phase. We find that neither the disordered N-terminal residues nor the structured C-terminal residues of the kinetically stable CFA-synthase dimer are required for FtsH recognition and degradation. Experiments with fusion proteins support a model in which an internal degron mediates FtsH recognition as a prelude to unfolding and proteolysis. These findings elucidate the terminal step in the life cycle of CFA synthase and provide new insight into FtsH function.

Keywords: AAA+ protease; ATP-dependent protein degradation; substrate recognition.

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • ATP-Dependent Proteases / chemistry
  • ATP-Dependent Proteases / metabolism
  • Bacterial Proteins / metabolism
  • Escherichia coli Proteins* / metabolism
  • Escherichia coli* / genetics
  • Escherichia coli* / metabolism
  • Proteolysis

Substances

  • Escherichia coli Proteins
  • cyclopropane synthetase
  • ATP-Dependent Proteases
  • Bacterial Proteins
  • FtsH protein, E coli