Attenuation of LPS-Induced Lung Injury by Benziodarone via Reactive Oxygen Species Reduction

Int J Mol Sci. 2023 Jun 12;24(12):10035. doi: 10.3390/ijms241210035.

Abstract

As overproduction of reactive oxygen species (ROS) causes various diseases, antioxidants that scavenge ROS, or inhibitors that suppress excessive ROS generation, can be used as therapeutic agents. From a library of approved drugs, we screened compounds that reduced superoxide anions produced by pyocyanin-stimulated leukemia cells and identified benzbromarone. Further investigation of several of its analogues showed that benziodarone possessed the highest activity in reducing superoxide anions without causing cytotoxicity. In contrast, in a cell-free assay, benziodarone induced only a minimal decrease in superoxide anion levels generated by xanthine oxidase. These results suggest that benziodarone is an inhibitor of NADPH oxidases in the plasma membrane but is not a superoxide anion scavenger. We investigated the preventive effect of benziodarone on lipopolysaccharide (LPS)-induced murine lung injury as a model of acute respiratory distress syndrome (ARDS). Intratracheal administration of benziodarone attenuated tissue damage and inflammation via its ROS-reducing activity. These results indicate the potential application of benziodarone as a therapeutic agent against diseases caused by ROS overproduction.

Keywords: ARDS; ROS; benzbromarone; benziodarone; superoxide anions.

MeSH terms

  • Animals
  • Humans
  • Lipopolysaccharides / toxicity
  • Lung Injury*
  • Mice
  • NADPH Oxidases / metabolism
  • Reactive Oxygen Species / metabolism
  • Superoxides

Substances

  • Reactive Oxygen Species
  • Superoxides
  • Lipopolysaccharides
  • benziodarone
  • NADPH Oxidases

Grants and funding

This research received no external funding.