Combined cytotoxic and immune-stimulatory gene therapy using Ad-TK and Ad-Flt3L: Translational developments from rodents to glioma patients

Mol Ther. 2023 Oct 4;31(10):2839-2860. doi: 10.1016/j.ymthe.2023.08.009. Epub 2023 Aug 12.

Abstract

Gliomas are the most prevalent and devastating primary malignant brain tumors in adults. Despite substantial advances in understanding glioma biology, there have been no regulatory drug approvals in the US since bevacizumab in 2009 and tumor treating fields in 2011. Recent phase III clinical trials have failed to meet their prespecified therapeutic primary endpoints, highlighting the need for novel therapies. The poor prognosis of glioma patients, resistance to chemo-radiotherapy, and the immunosuppressive tumor microenvironment underscore the need for the development of novel therapies. Gene therapy-based immunotherapeutic strategies that couple the ability of the host immune system to specifically kill glioma cells and develop immunological memory have shown remarkable progress. Two adenoviral vectors expressing Ad-HSV1-TK/GCV and Ad-Flt3L have shown promising preclinical data, leading to FDA approval of a non-randomized, phase I open-label, first in human trial to test safety, cytotoxicity, and immune-stimulatory efficiency in high-grade glioma patients (NCT01811992). This review provides a thorough overview of immune-stimulatory gene therapy highlighting recent advancements, potential drawbacks, future directions, and recommendations for future implementation of clinical trials.

Keywords: Ad-hCMV-TK + Ad-hCMV-Flt3L; dexamethasone; gene therapy; glioma; immune checkpoint inhibitors; immune-suppression; immunogenic cell death; oncolytic virotherapy; suicide gene therapy.

Publication types

  • Review

MeSH terms

  • Adenoviridae / genetics
  • Animals
  • Brain Neoplasms* / genetics
  • Brain Neoplasms* / pathology
  • Brain Neoplasms* / therapy
  • Genetic Therapy
  • Genetic Vectors / genetics
  • Glioma* / genetics
  • Glioma* / pathology
  • Glioma* / therapy
  • Humans
  • Rodentia / genetics
  • Thymidine Kinase / genetics
  • Tumor Microenvironment

Substances

  • Thymidine Kinase