Identification of drug candidates targeting monocyte reprogramming in people living with HIV

Front Immunol. 2023 Nov 20:14:1275136. doi: 10.3389/fimmu.2023.1275136. eCollection 2023.

Abstract

Introduction: People living with HIV (PLHIV) are characterized by functional reprogramming of innate immune cells even after long-term antiretroviral therapy (ART). In order to assess technical feasibility of omics technologies for application to larger cohorts, we compared multiple omics data layers.

Methods: Bulk and single-cell transcriptomics, flow cytometry, proteomics, chromatin landscape analysis by ATAC-seq as well as ex vivo drug stimulation were performed in a small number of blood samples derived from PLHIV and healthy controls from the 200-HIV cohort study.

Results: Single-cell RNA-seq analysis revealed that most immune cells in peripheral blood of PLHIV are altered in their transcriptomes and that a specific functional monocyte state previously described in acute HIV infection is still existing in PLHIV while other monocyte cell states are only occurring acute infection. Further, a reverse transcriptome approach on a rather small number of PLHIV was sufficient to identify drug candidates for reversing the transcriptional phenotype of monocytes in PLHIV.

Discussion: These scientific findings and technological advancements for clinical application of single-cell transcriptomics form the basis for the larger 2000-HIV multicenter cohort study on PLHIV, for which a combination of bulk and single-cell transcriptomics will be included as the leading technology to determine disease endotypes in PLHIV and to predict disease trajectories and outcomes.

Keywords: HIV; drug repurposing; inflammation; monocytes; systems immunology; transcriptomics.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Anti-HIV Agents* / therapeutic use
  • Cohort Studies
  • HIV Infections*
  • Humans
  • Monocytes
  • Multicenter Studies as Topic

Substances

  • Anti-HIV Agents

Grants and funding

The author(s) declare financial support was received for the research, authorship, and/or publication of this article. This work was supported by the German Research Foundation (DFG) (INST 37/1049-1, INST 216/981-1, INST 257/605-1, INST 269/768-1, INST 217/988-1, INST 217/577-1, EXC2151 – 390873048; and SFB1454 – 432325352 to JS); Helmholtz-Gemeinschaft Deutscher Forschungszentren, Germany (sparse2big to JS and MN); EU projects SYSCID (733100 to JS); ERA CVD (00160389 to JS); ImmunoSep (847422 to JS); AA is supported by DFG under AS 637/1-1; AS 637/2-1; AS 637/3-1 and SFB1454 (432325352) and by the BMBF under IMMME/01EJ2204D. The 2000-HIV study is supported by an unrestricted grant from ViiV Healthcare (to AV, M.G.N., LJ). MN is supported by an ERC Advanced Grant (833247) and a Spinoza Grant of the Netherlands Organization for Scientific Research.