Regulation of m6A Methylome in Cancer: Mechanisms, Implications, and Therapeutic Strategies

Cells. 2023 Dec 28;13(1):66. doi: 10.3390/cells13010066.

Abstract

Reversible N6-adenosine methylation of mRNA, referred to as m6A modification, has emerged as an important regulator of post-transcriptional RNA processing. Numerous studies have highlighted its crucial role in the pathogenesis of diverse diseases, particularly cancer. Post-translational modifications of m6A-related proteins play a fundamental role in regulating the m6A methylome, thereby influencing the fate of m6A-methylated RNA. A comprehensive understanding of the mechanisms that regulate m6A-related proteins and the factors contributing to the specificity of m6A deposition has the potential to unveil novel therapeutic strategies for cancer treatment. This review provides an in-depth overview of our current knowledge of post-translational modifications of m6A-related proteins, associated signaling pathways, and the mechanisms that drive the specificity of m6A modifications. Additionally, we explored the role of m6A-dependent mechanisms in the progression of various human cancers. Together, this review summarizes the mechanisms underlying the regulation of the m6A methylome to provide insight into its potential as a novel therapeutic strategy for the treatment of cancer.

Keywords: N6-methyladenosine (m6A) modification; m6A specificity; m6A-related proteins; novel therapeutic targets; post-translational modification.

Publication types

  • Review
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adenosine
  • Epigenome*
  • Humans
  • Knowledge
  • Neoplasms* / genetics
  • Neoplasms* / therapy
  • Protein Processing, Post-Translational

Substances

  • Adenosine

Grants and funding

This work was supported by a research grant from Chosun University (2023).