Analysis of autophagy in DLBCL reveals subtype-specific differences and the preferential targeting of ULK1 inhibition in GCB-DLBCL provides a rationale as a new therapeutic approach

Leukemia. 2024 Feb;38(2):424-429. doi: 10.1038/s41375-024-02147-4. Epub 2024 Jan 23.
No abstract available

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Autophagy
  • Autophagy-Related Protein-1 Homolog
  • Germinal Center
  • Humans
  • Intracellular Signaling Peptides and Proteins
  • Lymphoma, Large B-Cell, Diffuse* / drug therapy
  • Prognosis

Substances

  • ULK1 protein, human
  • Autophagy-Related Protein-1 Homolog
  • Intracellular Signaling Peptides and Proteins