[Mechanism of traditional Chinese medicine in treating ulcerative colitis-related colorectal cancer by regulating NF-κB-related signaling pathways: a review]

Zhongguo Zhong Yao Za Zhi. 2024 Mar;49(6):1455-1466. doi: 10.19540/j.cnki.cjcmm.20231222.601.
[Article in Chinese]

Abstract

Ulcerative colitis is a chronic, recurrent, and nonspecific intestinal inflammatory disease, which is difficult to cure and has the risk of deterioration into related tumors. Long-term chronic inflammatory stimulation can increase the risk of cancerization. With the signaling pathway as a key link in the regulation of tumor microenvironments, nuclear factor-kappa B(NF-κB) is an important regulator of intestinal inflammation. It can also be co-regulated as downstream factors of other signaling pathways, such as TLR4, MAPK, STAT, PI3K, and so on. At present, a large number of animal experiments have proved that traditional Chinese medicine(TCM) can reduce inflammation by interfering with NF-κB-related signaling pathways, improve intestinal inflammation, and inhibit the progression of inflammation to tumors. This article reviewed the relationship between NF-κB-related signaling pathways and the intervention mechanism of TCM, so as to provide a reference for the clinical treatment of ulcerative colitis and the optimization of related cancer prevention strategies.

Keywords: NF-κB; UCRCC; traditional Chinese medicine; ulcerative colitis.

Publication types

  • Review
  • English Abstract

MeSH terms

  • Animals
  • Colitis, Ulcerative* / complications
  • Colitis, Ulcerative* / drug therapy
  • Colitis, Ulcerative* / genetics
  • Colorectal Neoplasms* / drug therapy
  • Colorectal Neoplasms* / genetics
  • Disease Models, Animal
  • Inflammation
  • Medicine, Chinese Traditional
  • NF-kappa B / genetics
  • NF-kappa B / metabolism
  • Signal Transduction
  • Tumor Microenvironment

Substances

  • NF-kappa B