Association between STAT4 gene polymorphism and susceptibility to pulmonary tuberculosis in the Moldavian population

Int J Immunogenet. 2024 Aug;51(4):228-234. doi: 10.1111/iji.12672. Epub 2024 Apr 23.

Abstract

Signal transducer and activator of transcription 4 (STAT4) plays a crucial role in the host immune response against Mycobacterium tuberculosis. This study investigates the association between STAT4 gene polymorphisms and pulmonary tuberculosis (TB) risk in the Moldavian population. A total of 272 TB patients and 251 community-matched controls underwent screening for functional single-nucleotide polymorphisms (SNPs) rs897200 and rs7574865 in the STAT4 gene. The minor T allele and the TT/CT genotype of rs897200 demonstrated a significant association with reduced pulmonary TB risk (allelic model: adjusted OR = .74, p = .025; log-additive model: adjusted OR = .72, p = .02; and dominant model: adjusted OR = .65, p = .023), indicating a protective effect. Similar associations, characterized by an even more pronounced reduction in risk, were observed among females and late-onset TB patients (>44 years). No significant associations were found for rs7574865. In addition, a combined genotype analysis incorporating 43 SNPs from our previous studies revealed potential associations, such as STAT4 rs897200 CT with IFNG rs2430561 AA (adjusted OR = .36, p = .0025) and STAT4 rs897200 CT with TNFA rs1800629 GA (adjusted OR = .33, p = .0012). This study emphasizes the significant association of STAT4 rs897200 with pulmonary TB risk in the Moldavian population, underscoring its role in the disease development.

Keywords: association study; genetic susceptibility; host immunity; pulmonary tuberculosis; signal transducer and activator of transcription 4; single‐nucleotide polymorphism.

MeSH terms

  • Adult
  • Alleles
  • Case-Control Studies
  • Female
  • Gene Frequency
  • Genetic Association Studies
  • Genetic Predisposition to Disease*
  • Genotype
  • Humans
  • Male
  • Middle Aged
  • Moldova
  • Mycobacterium tuberculosis
  • Polymorphism, Single Nucleotide*
  • STAT4 Transcription Factor* / genetics
  • Tuberculosis, Pulmonary* / genetics

Substances

  • STAT4 Transcription Factor
  • STAT4 protein, human