Fluoxetine and Sertraline Potently Neutralize the Replication of Distinct SARS-CoV-2 Variants

Viruses. 2024 Mar 30;16(4):545. doi: 10.3390/v16040545.

Abstract

The pandemic caused by SARS-CoV-2 is still a major health problem. Newly emerging variants and long-COVID-19 represent a challenge for the global health system. In particular, individuals in developing countries with insufficient health care need easily accessible, affordable and effective treatments of COVID-19. Previous studies have demonstrated the efficacy of functional inhibitors of acid sphingomyelinase against infections with various viruses, including early variants of SARS-CoV-2. This work investigated whether the acid sphingomyelinase inhibitors fluoxetine and sertraline, usually used as antidepressant molecules in clinical practice, can inhibit the replication of the former and recently emerged SARS-CoV-2 variants in vitro. Fluoxetine and sertraline potently inhibited the infection with pseudotyped virus-like particles and SARS-CoV-2 variants D614G, alpha, delta, omicron BA.1 and omicron BA.5. These results highlight fluoxetine and sertraline as priority candidates for large-scale phase 3 clinical trials at different stages of SARS-CoV-2 infections, either alone or in combination with other medications.

Keywords: COVID-19; SARS-CoV-2; antidepressants.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antiviral Agents* / pharmacology
  • COVID-19 Drug Treatment
  • COVID-19* / virology
  • Chlorocebus aethiops
  • Fluoxetine* / pharmacology
  • Humans
  • SARS-CoV-2* / drug effects
  • Sertraline* / pharmacology
  • Vero Cells
  • Virus Replication* / drug effects

Substances

  • Sertraline
  • Fluoxetine
  • Antiviral Agents

Supplementary concepts

  • SARS-CoV-2 variants

Grants and funding

This study was supported by the Stiftung Universitätsmedizin Essen (awarded to A. Krawczyk), the Rudolf Ackermann Foundation (awarded to O. Witzke) and the German Research Foundation (DFG, funding number: KR 4476/5-1, awarded to A. Krawczyk, E. Gulbins, K. A. Becker(-Flegler) and M. Kamler). The authors acknowledge support from the Open Access Publication Fund of the University of Duisburg-Essen.