Emerging cell and molecular targets for treating mucus hypersecretion in asthma

Allergol Int. 2024 Jul;73(3):375-381. doi: 10.1016/j.alit.2024.04.002. Epub 2024 May 1.

Abstract

Mucus provides a protective barrier that is crucial for host defense in the lungs. However, excessive or abnormal mucus can have pathophysiological consequences in many pulmonary diseases, including asthma. Patients with asthma are treated with agents that relax airway smooth muscle and reduce airway inflammation, but responses are often inadequate. In part, this is due to the inability of existing therapeutic agents to directly target mucus. Accordingly, there is a critical need to better understand how mucus hypersecretion and airway plugging are affected by the epithelial cells that synthesize, secrete, and transport mucus components. This review highlights recent advances in the biology of mucin glycoproteins with a specific focus on MUC5AC and MUC5B, the chief macromolecular components of airway mucus. An improved mechanistic understanding of key steps in mucin production and secretion will help reveal novel potential therapeutic strategies.

Keywords: Asthma; Mucin; Mucous cell; Mucus; Secretion.

Publication types

  • Review

MeSH terms

  • Animals
  • Asthma* / drug therapy
  • Asthma* / metabolism
  • Humans
  • Molecular Targeted Therapy
  • Mucin 5AC / metabolism
  • Mucin-5B / metabolism
  • Mucins / metabolism
  • Mucus* / metabolism
  • Respiratory Mucosa / immunology
  • Respiratory Mucosa / metabolism

Substances

  • Mucins
  • Mucin 5AC
  • Mucin-5B