Mesolimbic dopaminergic neurons are not spared by MPTP neurotoxicity in mice

Eur J Pharmacol. 1985 Aug 7;114(1):97-100. doi: 10.1016/0014-2999(85)90528-x.

Abstract

In C57 black mice, MPTP (N-methyl-4-phenyl-1,2,3,6-tetrahydropyridine) (30 mg/kg X 2, 20 mg/kg X 15, 30 mg/kg X 15, or 35 mg/kg X 1) depleted DA in both striatum and accumbens but not in frontal cortex, hypothalamus and retina. DA decreases were more pronounced in striatum than in accumbens, were maximal at 2 days, partially reversed later but persisted up to 30 days after treatment. DA depletions in nigra were smaller earlier and maximal later. The study suggests that, in mice, MPTP damages nigrostriatal and mesolimbic projections but spares other DA neurons.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine
  • Animals
  • Cerebral Cortex / metabolism
  • Corpus Striatum / metabolism
  • Dopamine / metabolism
  • Dopamine / physiology*
  • Hypothalamus / metabolism
  • Limbic System / drug effects*
  • Male
  • Mice
  • Mice, Inbred C57BL
  • Neurons / drug effects*
  • Nucleus Accumbens / metabolism
  • Pyridines / toxicity*
  • Retina / metabolism
  • Substantia Nigra / metabolism
  • Time Factors

Substances

  • Pyridines
  • 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine
  • Dopamine