PI3KC2α controls cardiac contractility through regulation of β2-adrenergic receptor recycling

Vascul Pharmacol. 2024 Jun:155:107313. doi: 10.1016/j.vph.2024.107313.

Affiliations

¹ Department of Molecular Biotechnology and Health Sciences, Molecular Biotechnology Center, University of Torino, 10126 Torino, Italy.
² Institute of Pharmacology and Toxicology, University of Würzburg, 97078 Würzburg, Germany.
³ Institute of Pharmacology and Toxicology, University of Würzburg, 97078 Würzburg, Germany; Leibniz-Institut für Analytische Wissenschaften - ISAS - e.V., 44139 Dortmund, Germany.

PMID: 38985607
DOI: 10.1016/j.vph.2024.107313

No abstract available

MeSH terms

Animals
Class I Phosphatidylinositol 3-Kinases / genetics
Class I Phosphatidylinositol 3-Kinases / metabolism
Humans
Myocardial Contraction*
Myocytes, Cardiac* / enzymology
Myocytes, Cardiac* / metabolism
Receptors, Adrenergic, beta-2* / genetics
Receptors, Adrenergic, beta-2* / metabolism
Signal Transduction

Substances

Receptors, Adrenergic, beta-2
Class I Phosphatidylinositol 3-Kinases