Cellular mechanisms in selected states of insulin resistance: human obesity, glucocorticoid excess, and chronic renal failure

Diabetes Metab Rev. 1985;1(3):293-317. doi: 10.1002/dmr.5610010304.
No abstract available

Publication types

  • Research Support, U.S. Gov't, P.H.S.
  • Review

MeSH terms

  • Animals
  • Glucocorticoids / metabolism*
  • Glucose / metabolism
  • Humans
  • Insulin Resistance*
  • Intercellular Signaling Peptides and Proteins
  • Lipids / biosynthesis
  • Liver / metabolism
  • Liver Glycogen / biosynthesis
  • Obesity / metabolism*
  • Peptides / physiology
  • Receptor, Insulin / metabolism
  • Uremia / metabolism*

Substances

  • Glucocorticoids
  • Intercellular Signaling Peptides and Proteins
  • Lipids
  • Liver Glycogen
  • Peptides
  • insulin resistance factor (uremia)
  • Receptor, Insulin
  • Glucose