The NOTCH1 and miR-34a signaling network is affected by TP53 alterations in CLL

Leuk Lymphoma. 2024 Dec;65(13):1941-1953. doi: 10.1080/10428194.2024.2392839. Epub 2024 Aug 19.

Abstract

In chronic lymphocytic leukemia (CLL), TP53 mutations or deletions on chromosome 17p lead to adverse prognosis and reduced levels of miR-34a, which targets NOTCH1. Also, hyperactivated NOTCH1 signaling is crucial for CLL progression. Here we explored the interaction between p53, miR-34a, and NOTCH1 in CLL. We investigated the effect of p53 and miR-34a on NOTCH1 signaling and expression in CLL cells with altered TP53. Our results indicate that miR-34a reduces NOTCH1 3' UTR activity but might not be a mediator between p53 signaling and NOTCH1. p53 activation increases miR-34a expression and NOTCH1 protein levels, correlating with decreased NOTCH1 and miR-34a levels in primary CLL cells with TP53 alterations. Some samples with high NOTCH1 levels presented increased BCL-2, suggesting an anti-apoptotic mechanism of a potentially direct p53-NOTCH1 relation in CLL. This study deepens the understanding of the p53-miR-34a-NOTCH1 signaling network, providing insights that could guide future therapeutic strategies for CLL.

Keywords: CLL; NOTCH1; miR-34a; p53; signaling network.

MeSH terms

  • 3' Untranslated Regions
  • Apoptosis / genetics
  • Cell Line, Tumor
  • Gene Expression Regulation, Leukemic
  • Humans
  • Leukemia, Lymphocytic, Chronic, B-Cell* / genetics
  • Leukemia, Lymphocytic, Chronic, B-Cell* / metabolism
  • Leukemia, Lymphocytic, Chronic, B-Cell* / pathology
  • MicroRNAs* / genetics
  • Mutation
  • RNA Interference
  • Receptor, Notch1* / genetics
  • Receptor, Notch1* / metabolism
  • Signal Transduction*
  • Tumor Suppressor Protein p53* / genetics
  • Tumor Suppressor Protein p53* / metabolism

Substances

  • MicroRNAs
  • Receptor, Notch1
  • Tumor Suppressor Protein p53
  • MIRN34 microRNA, human
  • NOTCH1 protein, human
  • 3' Untranslated Regions
  • TP53 protein, human