Glutathione transferase omega 1-1 (GSTO1-1) can effect the inter-cell transfer of cisplatin resistance through the exosomal route

Free Radic Biol Med. 2024 Nov 1:224:162-167. doi: 10.1016/j.freeradbiomed.2024.08.032. Epub 2024 Aug 27.

Abstract

Glutathione transferase omega-1-1 (GSTO1-1) is a member of the glutathione transferase superfamily (GSTs) involved in the modulation of cell survival, proliferation and metabolism. Increased levels of GSTO1-1 have been associated with cancer progression and chemoresistance in different types of cancer cells, possibly supported by the post-traslational regulation of some major prosurvival pathways regulated by the enzyme. Our data demonstrate for the first time that GSTO1-1 can be released by cancer cells through the exosomal route and transferred to GSTO1-1 knock-out cells, this resulting in an increased resistance against cisplatin toxicity in recipient cells. The use of the exosomal route to transfer the regulatory competences of GSTO1-1 could be a further element supporting its role in neoplastic progression.

Keywords: Cisplatin; Exosomes; Glutathione transferase omega-1-1.

MeSH terms

  • Antineoplastic Agents / pharmacology
  • Cell Line, Tumor
  • Cell Proliferation / drug effects
  • Cell Survival / drug effects
  • Cisplatin* / pharmacology
  • Drug Resistance, Neoplasm* / genetics
  • Exosomes* / genetics
  • Exosomes* / metabolism
  • Glutathione Transferase* / genetics
  • Glutathione Transferase* / metabolism
  • Humans
  • Neoplasms / drug therapy
  • Neoplasms / genetics
  • Neoplasms / metabolism
  • Neoplasms / pathology

Substances

  • Cisplatin
  • Glutathione Transferase
  • GSTO1 protein, human
  • Antineoplastic Agents