Long-term survival of kidney-transplant recipient with donor-transmitted malignant melanoma after provoked rejection

Transpl Immunol. 2024 Dec:87:102117. doi: 10.1016/j.trim.2024.102117. Epub 2024 Sep 2.

Abstract

Donor-transmitted malignancy is a rare and often fatal complication of organ transplantation. We report a case of a 55-year old male kidney transplant recipient who was diagnosed with stage-IV donor-transmitted melanoma 5 months after transplantation with metastases in the liver, spleen, lung, and brain. Immunosuppression was discontinued, and encorafenib and binimetinib, inhibitors of a serine/threonine B-Raf proto-oncogene (BRAF) and mitogen-activated protein kinase kinase (MEK) respectively, were started. Severe rejection ensued and necessitated the start of hemodialysis as well as urgent graft nephrectomy. However, the tumor progressed and BRAF/MEK inhibition was replaced by immune-checkpoint inhibition with ipilimumab and nivolumab. When this also failed to slow disease progression and seizures occurred, therapy with encorafenib and binimetinib was reinstated. Afterwards, most of the metastases remained stable. The patient has now survived for more than 4 years in good general health, which is an exceptionally long survival with donor-transmitted, metastasized melanoma.

Keywords: Donor transmitted malignancy; Donor transmitted melanoma; Melanoma kidney transplant; Transplantation malignancy.

Publication types

  • Case Reports

MeSH terms

  • Benzimidazoles / therapeutic use
  • Carbamates / therapeutic use
  • Graft Rejection*
  • Humans
  • Kidney Transplantation*
  • Male
  • Melanoma*
  • Middle Aged
  • Proto-Oncogene Mas*
  • Proto-Oncogene Proteins B-raf / antagonists & inhibitors
  • Proto-Oncogene Proteins B-raf / genetics
  • Skin Neoplasms
  • Sulfonamides / therapeutic use
  • Tissue Donors*
  • Transplant Recipients

Substances

  • Proto-Oncogene Mas
  • MAS1 protein, human
  • Carbamates
  • encorafenib
  • Benzimidazoles
  • Proto-Oncogene Proteins B-raf
  • Sulfonamides
  • binimetinib
  • BRAF protein, human