Paclitaxel hyperthermia suppresses gastric cancer migration through MiR-183-5p/PPP2CA/AKT/GSK3β/β-catenin axis

J Cancer Res Clin Oncol. 2024 Sep 9;150(9):416. doi: 10.1007/s00432-024-05923-y.

Abstract

Background: Gastric cancer (GC), a prevalent malignant tumor which is a leading cause of death from malignancy around the world. Peritoneal metastasis accounts for the major cause of mortality in patients with GC. Despite hyperthermia intraperitoneal chemotherapy (HIPEC) improves the therapeutic effect of GC, it's equivocal about the mechanism under HIPEC.

Methods: MiR-183-5p expression was sifted from miRNA chip and detected in both GC patients and cell lines by qRT-PCR. Gene interference and rescue experiments were performed to identified biological function in vitro and vivo. Next, we affirmed PPP2CA as targeted of miR-183-5p by dual luciferase reporter assay. Finally, the potential relationship between HIPEC and miR-183-5p was explored.

Results: MiR-183-5p is up-regulated in GC and associated with advanced stage and poor prognosis. MiR-183-5p accelerate GC migration in vitro which is influenced by miR-183-5p/PPP2CA/AKT/GSK3β/β-catenin Axis. HIPEC exerts migration inhibition via attenuating miR-183-5p expression.

Conclusion: MiR-183-5p can be used as a potential HIPEC biomarker in patients with CC.

Keywords: Gastric cancer; HIPEC; MiR-183-5p; Migration.

MeSH terms

  • Animals
  • Antineoplastic Agents, Phytogenic / pharmacology
  • Cell Line, Tumor
  • Cell Movement*
  • Female
  • Gene Expression Regulation, Neoplastic
  • Glycogen Synthase Kinase 3 beta* / genetics
  • Glycogen Synthase Kinase 3 beta* / metabolism
  • Humans
  • Hyperthermia, Induced* / methods
  • Male
  • Mice
  • Mice, Inbred BALB C
  • Mice, Nude
  • MicroRNAs* / genetics
  • Middle Aged
  • Prognosis
  • Protein Phosphatase 2 / genetics
  • Protein Phosphatase 2 / metabolism
  • Proto-Oncogene Proteins c-akt* / metabolism
  • Stomach Neoplasms* / genetics
  • Stomach Neoplasms* / metabolism
  • Stomach Neoplasms* / pathology
  • Xenograft Model Antitumor Assays
  • beta Catenin* / genetics
  • beta Catenin* / metabolism

Substances

  • MicroRNAs
  • Glycogen Synthase Kinase 3 beta
  • beta Catenin
  • Proto-Oncogene Proteins c-akt
  • MIRN183 microRNA, human
  • Protein Phosphatase 2
  • CTNNB1 protein, human
  • GSK3B protein, human
  • Antineoplastic Agents, Phytogenic