Time-dependent inhibition of the cyclooxygenase pathway by 12-hydroperoxy-5,8,10,14-eicosatetraenoic acid

Biochem Biophys Res Commun. 1985 Jul 31;130(2):781-5. doi: 10.1016/0006-291x(85)90484-x.

Abstract

We examined effects of small dose (1 microM or less) of exogenous 12-hydroperoxy-5,8,10,14-eicosatetraenoic acid (12-HPETE) on the formation of cyclooxygenase products from exogenous arachidonic acid (AA) in washed human platelets. With a simultaneous addition of AA, 12-HPETE did not affect the formation of thromboxane (TX)B2 and 12-hydroxy-5,8,10-heptadecatrienoic acid (HHT). However, by being preincubated with platelets before an addition of AA, 0.1 microM or greater of 12-HPETE inhibited the formation of TXB2 and HHT dose-dependently. In addition, the inhibitory effect of 12-HPETE increased as the preincubation time was prolonged. These results suggest that 12-HPETE is a strong inhibitor for the cyclooxygenase pathway.

MeSH terms

  • 12-Hydroxy-5,8,10,14-eicosatetraenoic Acid
  • Arachidonic Acid
  • Arachidonic Acids / metabolism
  • Arachidonic Acids / pharmacology*
  • Blood Platelets / drug effects
  • Blood Platelets / enzymology*
  • Cyclooxygenase Inhibitors*
  • Dose-Response Relationship, Drug
  • Humans
  • Hydroxyeicosatetraenoic Acids / biosynthesis
  • Leukotrienes*
  • Thromboxane B2 / biosynthesis
  • Time Factors

Substances

  • Arachidonic Acids
  • Cyclooxygenase Inhibitors
  • Hydroxyeicosatetraenoic Acids
  • Leukotrienes
  • Arachidonic Acid
  • Thromboxane B2
  • 12-Hydroxy-5,8,10,14-eicosatetraenoic Acid
  • 12-HPETE