Multiomic profiling identifies predictors of survival in African American patients with acute myeloid leukemia

Nat Genet. 2024 Nov;56(11):2434-2446. doi: 10.1038/s41588-024-01929-x. Epub 2024 Oct 4.

Abstract

Genomic profiles and prognostic biomarkers in patients with acute myeloid leukemia (AML) from ancestry-diverse populations are underexplored. We analyzed the exomes and transcriptomes of 100 patients with AML with genomically confirmed African ancestry (Black; Alliance) and compared their somatic mutation frequencies with those of 323 self-reported white patients with AML, 55% of whom had genomically confirmed European ancestry (white; BeatAML). Here we find that 73% of 162 gene mutations recurrent in Black patients, including a hitherto unreported PHIP alteration detected in 7% of patients, were found in one white patient or not detected. Black patients with myelodysplasia-related AML were younger than white patients suggesting intrinsic and/or extrinsic dysplasia-causing stressors. On multivariable analyses of Black patients, NPM1 and NRAS mutations were associated with inferior disease-free and IDH1 and IDH2 mutations with reduced overall survival. Inflammatory profiles, cell type distributions and transcriptional profiles differed between Black and white patients with NPM1 mutations. Incorporation of ancestry-specific risk markers into the 2022 European LeukemiaNet genetic risk stratification changed risk group assignment for one-third of Black patients and improved their outcome prediction.

MeSH terms

  • Adult
  • Aged
  • Biomarkers, Tumor / genetics
  • Black or African American* / genetics
  • Female
  • GTP Phosphohydrolases / genetics
  • Gene Expression Profiling
  • Humans
  • Isocitrate Dehydrogenase / genetics
  • Leukemia, Myeloid, Acute* / genetics
  • Leukemia, Myeloid, Acute* / mortality
  • Male
  • Membrane Proteins / genetics
  • Middle Aged
  • Mutation*
  • Nuclear Proteins / genetics
  • Nucleophosmin*
  • Prognosis
  • Transcriptome
  • White / genetics

Substances

  • Biomarkers, Tumor
  • GTP Phosphohydrolases
  • IDH1 protein, human
  • IDH2 protein, human
  • Isocitrate Dehydrogenase
  • Membrane Proteins
  • NPM1 protein, human
  • NRAS protein, human
  • Nuclear Proteins
  • Nucleophosmin