A Siah3 of relief: A role for mitophagy as a fail-safe during developmental axon pruning

Sci Signal. 2024 Oct 8;17(857):eads1228. doi: 10.1126/scisignal.ads1228. Epub 2024 Oct 8.

Abstract

Developmental axon pruning is controlled by a careful balance of pro- and anti-apoptotic signals, which are activated in response to external cues to sculpt mature neuronal circuitry. In this issue of Science Signaling, Abraham et al. define a safeguard against apoptotic axon pruning and illustrate that Siah3 represses Parkin-mediated mitophagy to control the availability of axonal mitochondria that activate the pruning process.

MeSH terms

  • Animals
  • Apoptosis
  • Axons* / metabolism
  • Axons* / physiology
  • Humans
  • Mice
  • Mitochondria* / metabolism
  • Mitophagy* / physiology
  • Nuclear Proteins / genetics
  • Nuclear Proteins / metabolism
  • Seven in Absentia Proteins
  • Ubiquitin-Protein Ligases* / genetics
  • Ubiquitin-Protein Ligases* / metabolism

Substances

  • Nuclear Proteins
  • parkin protein
  • Seven in Absentia Proteins
  • Ubiquitin-Protein Ligases