Trial watch: anticancer vaccination with dendritic cells

Oncoimmunology. 2024 Oct 9;13(1):2412876. doi: 10.1080/2162402X.2024.2412876. eCollection 2024.

Abstract

Dendritic cells (DCs) are critical players at the intersection of innate and adaptive immunity, making them ideal candidates for anticancer vaccine development. DC-based immunotherapies typically involve isolating patient-derived DCs, pulsing them with tumor-associated antigens (TAAs) or tumor-specific antigens (TSAs), and utilizing maturation cocktails to ensure their effective activation. These matured DCs are then reinfused to elicit tumor-specific T-cell responses. While this approach has demonstrated the ability to generate potent immune responses, its clinical efficacy has been limited due to the immunosuppressive tumor microenvironment. Recent efforts have focused on enhancing the immunogenicity of DC-based vaccines, particularly through combination therapies with T cell-targeting immunotherapies. This Trial Watch summarizes recent advances in DC-based cancer treatments, including the development of new preclinical and clinical strategies, and discusses the future potential of DC-based vaccines in the evolving landscape of immuno-oncology.

Keywords: Antigen cross-presentation; DAMPs; T cell priming; TAAs; clinical trial; dendritic cells; immune checkpoint blockers; tumor-infiltrating lymphocytes.

Publication types

  • Review

MeSH terms

  • Animals
  • Antigens, Neoplasm / immunology
  • Cancer Vaccines* / administration & dosage
  • Cancer Vaccines* / immunology
  • Clinical Trials as Topic
  • Dendritic Cells* / immunology
  • Humans
  • Immunotherapy / methods
  • Neoplasms* / immunology
  • Neoplasms* / therapy
  • Tumor Microenvironment / immunology
  • Vaccination / methods

Substances

  • Antigens, Neoplasm
  • Cancer Vaccines

Grants and funding

ADG is supported by Research Foundation Flanders (FWO) (Fundamental Research Grant, G0B4620N; Excellence of Science/EOS grant, 30837538, for ‘DECODE’ consortium; Strategic Basic Research or SBO grant, S000523N), KU Leuven (C1 grant, C14/24/122; and C3 grants, C3/23/067, C3/21/037, C3/22/022), Come Up Against Cancer or Kom op Tegen Kanker (KOTK/2018/11509/1 and KOTK/2019/11955/1), VLIR-UOS (iBOF grant, iBOF/21/048, for ‘MIMICRY’ consortium), Olivia Hendrickx Research Foundation (OHRF), and European Union (EU) Mission Cancer grant for the GLIOMATCH consortium (Project no. 101136670). IV and RSL are supported by FWO-SB PhD Fellowship (1S06821N and 1S44123N). JS was funded by Kom op tegen Kanker (Stand up to Cancer), the Flemish cancer society through the Emmanuel van der Schueren (EvDS) PhD fellowship (projectID: 12699).