Female patients exhibit altered vasopressin-induced coronary microvascular contractile response and molecular signaling following cardiac surgery

Am J Surg. 2025 Jan:239:116052. doi: 10.1016/j.amjsurg.2024.116052. Epub 2024 Oct 29.

Abstract

Background: Emerging data suggest women have worse outcomes than men following cardioplegia and cardiopulmonary bypass (CP/CPB). Altered coronary microvascular function affecting myocardial perfusion may contribute, but human translational studies are lacking.

Methods: Viable coronary microvessels (<200 μ m) were dissected from human atrial samples collected before and after CP/CPB from a subset of 108 patients enrolled. Ex vivo contractile responses to vasopressin were assessed using video microscopy. RNA deep-sequencing and immunoblotting were used to quantify gene and protein expression, respectively.

Results: Coronary microvessels exhibited increased vasopressin-induced contractile responses post-CP/CPB in males and females (p ​< ​0.0001). Females exhibited a decrease in microvascular contractile response versus males pre- (p ​= ​0.1) and post-CP/CPB (p ​= ​0.09) which approached significance. Myocardial vasopressin 1a receptor levels were increased in females versus males (p ​= ​0.001). Vasopressin-induced vasoconstriction predicted postoperative cardiac index.

Conclusions: Impaired coronary microvascular contractile responses in females jeopardizing myocardial perfusion may underlie worse outcomes following cardiac surgery.

Keywords: Coronary microvascular reactivity; Sex differences; Vasopressin.

MeSH terms

  • Aged
  • Cardiac Surgical Procedures / adverse effects
  • Cardiopulmonary Bypass / adverse effects
  • Coronary Circulation / drug effects
  • Coronary Vessels* / drug effects
  • Coronary Vessels* / metabolism
  • Female
  • Heart Arrest, Induced / adverse effects
  • Humans
  • Male
  • Microcirculation / drug effects
  • Microvessels / drug effects
  • Middle Aged
  • Receptors, Vasopressin / metabolism
  • Sex Factors
  • Signal Transduction
  • Vasoconstriction* / drug effects
  • Vasopressins* / pharmacology

Substances

  • Vasopressins
  • Receptors, Vasopressin