Structural basis of signaling complex inhibition by IL-6 domain-swapped dimers

Structure. 2025 Jan 2;33(1):171-180.e5. doi: 10.1016/j.str.2024.10.028. Epub 2024 Nov 19.

Abstract

Interleukin-6 (IL-6) is a multifaceted cytokine essential in many immune system processes and their regulation. It also plays a key role in hematopoiesis, and in triggering the acute phase reaction. IL-6 overproduction is critical in chronic inflammation associated with autoimmune diseases like rheumatoid arthritis and contributes to cytokine storms in COVID-19 patients. Over 20 years ago, researchers proposed that IL-6, which is typically monomeric, can also form dimers via a domain-swap mechanism, with indirect evidence supporting their existence. The physiological significance of IL-6 dimers was shown in B-cell chronic lymphocytic leukemia. However, no structures have been reported so far. Here, we present the crystal structure of an IL-6 domain-swapped dimer that computational approaches could not predict. The structure explains why the IL-6 dimer is antagonistic to the IL-6 monomer in signaling complex formation and provides insights for IL-6 targeted therapies.

Keywords: AlphaFold; IL-6; antibody; cytokine; dimerization; domain-swap; fragment antigen-binding; gp130; interleukin 6; structure.

MeSH terms

  • Binding Sites
  • Crystallography, X-Ray
  • Humans
  • Interleukin-6* / chemistry
  • Interleukin-6* / metabolism
  • Models, Molecular*
  • Protein Binding
  • Protein Domains
  • Protein Multimerization*
  • Signal Transduction*

Substances

  • Interleukin-6
  • IL6 protein, human