Integration of bulk/scRNA-seq and multiple machine learning algorithms identifies PIM1 as a biomarker associated with cuproptosis and ferroptosis in abdominal aortic aneurysm

Front Immunol. 2024 Dec 11:15:1486209. doi: 10.3389/fimmu.2024.1486209. eCollection 2024.

Abstract

Background: Abdominal aortic aneurysm (AAA) is a serious life-threatening vascular disease, and its ferroptosis/cuproptosis markers have not yet been characterized. This study was aiming to identify markers associated with ferroptosis/cuproptosis in AAA by bioinformatics analysis combined with machine learning models and to perform experimental validation.

Methods: This study used three scRNA-seq datasets from different mouse models and a human PBMC bulk RNA-seq dataset. Candidate genes were identified by integrated analysis of scRNA-seq, cell communication analysis, monocle pseudo-time analysis, and hdWGCNA analysis. Four machine learning algorithms, LASSO, REF, RF and SVM, were used to construct a prediction model for the PBMC dataset, the above results were comprehensively analyzed, and the targets were confirmed by RT-qPCR.

Results: scRNA-seq analysis showed Mo/MF as the most sensitive cell type to AAA, and 34 cuproptosis associated ferroptosis genes were obtained. Pseudo-time series analysis, hdWGCNA and machine learning prediction model construction were performed on these genes. Subsequent comparison of the above results showed that only PIM1 appeared in all algorithms. RT-qPCR and western blot results were consistent with sequencing results, showing that PIM1 was significantly upregulated in AAA.

Conclusion: In a conclusion, PIM1 as a novel biomarker associated with cuproptosis/ferroptosis in AAA was highlighted.

Keywords: PIM1; WGCNA; abdominal aortic aneurysm; cuproptosis; ferroptosis.

MeSH terms

  • Algorithms
  • Animals
  • Aortic Aneurysm, Abdominal* / genetics
  • Biomarkers*
  • Computational Biology / methods
  • Disease Models, Animal
  • Ferroptosis* / genetics
  • Gene Expression Profiling
  • Humans
  • Machine Learning*
  • Male
  • Mice
  • Proto-Oncogene Proteins c-pim-1* / genetics
  • Proto-Oncogene Proteins c-pim-1* / metabolism
  • RNA-Seq
  • Single-Cell Gene Expression Analysis

Substances

  • Biomarkers
  • Proto-Oncogene Proteins c-pim-1
  • PIM1 protein, human

Grants and funding

The author(s) declare financial support was received for the research, authorship, and/or publication of this article. This work was supported by the Shandong postdoctoral innovation project (SDCX-ZG-202303087).