Invasion of glioma cells through confined space requires membrane tension regulation and mechano-electrical coupling via Plexin-B2

Nat Commun. 2025 Jan 2;16(1):272. doi: 10.1038/s41467-024-55056-6.

Abstract

Glioblastoma (GBM) is a malignant brain tumor with diffuse infiltration. Here, we demonstrate how GBM cells usurp guidance receptor Plexin-B2 for confined migration through restricted space. Using live-cell imaging to track GBM cells negotiating microchannels, we reveal endocytic vesicle accumulation at cell front and filamentous actin assembly at cell rear in a polarized manner. These processes are interconnected and require Plexin-B2 signaling. We further show that Plexin-B2 governs membrane tension and other membrane features such as endocytosis, phospholipid composition, and inner leaflet surface charge, thus providing biophysical mechanisms by which Plexin-B2 promotes GBM invasion. Together, our studies unveil how GBM cells regulate membrane tension and mechano-electrical coupling to adapt to physical constraints and achieve polarized confined migration.

MeSH terms

  • Actins / metabolism
  • Brain Neoplasms* / metabolism
  • Brain Neoplasms* / pathology
  • Cell Line, Tumor
  • Cell Membrane* / metabolism
  • Cell Movement* / physiology
  • Endocytosis / physiology
  • Glioblastoma* / metabolism
  • Glioblastoma* / pathology
  • Glioma / metabolism
  • Glioma / pathology
  • Humans
  • Neoplasm Invasiveness*
  • Nerve Tissue Proteins* / genetics
  • Nerve Tissue Proteins* / metabolism
  • Signal Transduction

Substances

  • Nerve Tissue Proteins
  • PLXNB2 protein, human
  • Actins