Regulation of myosin light chain kinase by reversible phosphorylation and calcium-calmodulin

Ann N Y Acad Sci. 1980:356:142-50. doi: 10.1111/j.1749-6632.1980.tb29607.x.

Abstract

1) Myosin light chain kinases from smooth muscle and platelets can be phosphorylated by the catalytic subunit of cAMP-dependent protein kinase. 2) Phosphorylation of both kinases, in the absence of calmodulin, markedly decreases kinase activity. 3) The decrease in smooth muscle myosin kinase activity is due to a decreased affinity of the phosphorylated kinase for calmodulin. 4) Dephosphorylation of the smooth muscle kinase by a phosphatase isolated from smooth muscle restores the affinity of the kinase for calmodulin.

MeSH terms

  • Animals
  • Blood Platelets / enzymology
  • Calcium / pharmacology*
  • Calcium-Binding Proteins / pharmacology*
  • Calmodulin / pharmacology*
  • Gizzard, Avian / enzymology
  • Humans
  • Kinetics
  • Myosin-Light-Chain Kinase
  • Myosin-Light-Chain Phosphatase
  • Phosphoprotein Phosphatases / metabolism
  • Phosphorylation
  • Protein Kinases / metabolism*
  • Turkeys

Substances

  • Calcium-Binding Proteins
  • Calmodulin
  • Protein Kinases
  • Myosin-Light-Chain Kinase
  • Phosphoprotein Phosphatases
  • Myosin-Light-Chain Phosphatase
  • Calcium