Inhibition of peptidyltransferase and possible mode of action of a dipeptidyl chloramphenicol analog

Biochem Biophys Res Commun. 1984 Jul 31;122(2):748-54. doi: 10.1016/s0006-291x(84)80097-2.

Abstract

A dipeptidyl chloramphenicol analog, D-threo-2-(L-phenylalanylglycyl)amino-3-p-nitrophenyl-1,3- propanediol, has been prepared and examined as an inhibitor of ribosomal peptidyltransferase. The analog is a more effective inhibitor of poly (U,C) directed protein biosynthesis in an Escherichia coli cell-free system than chloramphenicol and shows inhibitory activity equal to the parent antibiotic in the transpeptidation reaction. These results and the common structural features of puromycin and this compound suggest a model for the binding modes of chloramphenicol and chloramphenicol analogs. This proposal invokes four major binding pockets at the A-site of the peptidyltransferase center.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Acyltransferases / antagonists & inhibitors*
  • Binding Sites
  • Chloramphenicol / analogs & derivatives*
  • Chloramphenicol / pharmacology
  • Escherichia coli / enzymology*
  • Kinetics
  • Peptidyl Transferases / antagonists & inhibitors*
  • Puromycin / pharmacology
  • RNA, Transfer, Amino Acyl / metabolism
  • Ribosomes / enzymology
  • Structure-Activity Relationship

Substances

  • RNA, Transfer, Amino Acyl
  • Puromycin
  • Chloramphenicol
  • 2-(phenylalanylglycyl)amino-3-(4-nitrophenyl)-1,3-propanediol
  • Acyltransferases
  • Peptidyl Transferases