High pO2-activated inhibitor of protein synthesis in rabbit reticulocytes: its relationship to glutathione disulfide-induced inhibitor and to a approximately 23,000-Mr sulfhydryl protein

Biochem Biophys Res Commun. 1983 Nov 30;117(1):135-40. doi: 10.1016/0006-291x(83)91551-6.

Abstract

The treatment of reticulocyte post-ribosomal supernatant containing ribosome wash with high pO2 or glutathione disulfide resulted in the activation of an inhibitor of protein synthesis of approximately 23,000-Mr as implicated by its elution from Sephadex G-100. This inhibitor could also be directly activated by exposure of the approximately 23,000-Mr fractions of the control eluate to high pO2 or glutathione disulfide. The high pO2-dependent activation of the inhibitor was blocked by the presence of glucose-6-phosphate or cAMP (2 mM). The inhibitor was stable (and activable) during a 5 minute incubation at 80 degrees C. The analysis by sodium dodecyl sulfate-polyacrylamide gel electrophoresis of the G-100 (approximately 23,000-Mr) fractions treated with [14C]N-ethylmaleimide revealed the abolishment of the label in a approximately 23,000-Mr protein band in parallel to high pO2-dependent inhibitor activation.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Ethylmaleimide / metabolism
  • Glutathione / analogs & derivatives
  • Glutathione / pharmacology
  • Glutathione Disulfide
  • Hot Temperature
  • Oxygen / blood*
  • Partial Pressure
  • Protein Biosynthesis* / drug effects
  • Proteins / antagonists & inhibitors
  • Proteins / isolation & purification
  • Rabbits
  • Reticulocytes / drug effects
  • Reticulocytes / metabolism*
  • Ribosomes / metabolism
  • Sulfhydryl Compounds / isolation & purification
  • Sulfhydryl Compounds / metabolism

Substances

  • Proteins
  • Sulfhydryl Compounds
  • Glutathione
  • Ethylmaleimide
  • Oxygen
  • Glutathione Disulfide