Aging and the regulation of striatal dopaminergic mechanisms in mice

J Pharmacol Exp Ther. 1981 Dec;219(3):695-700.

Abstract

Compensatory responses of striatal dopaminergic mechanisms to chronic drug treatment were tested in C57BL/6J and C57BL/6NNia male mice during the adult lifespan (5, 12, and 24 to 26 months). Chronic treatment with haloperidol (dopamine antagonist) (1.2 or 2 mg/kg/day for 21 days) induced supersensitivity in younger mice (5 and 12 months) as monitored by increased striatal spiroperidol binding sites and increased intensity of apomorphine-induced stereotyped behavior; in contrast, old mice were unaffected. However, chronic treatment with bromocriptine (dopamine agonist) (15 mg/kg/day for 7 days) induced desensitization at all ages, as monitored by reduced spiroperidol binding sites and reduced intensity of stereotyped behavior.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Aging*
  • Animals
  • Apomorphine / pharmacology
  • Bromocriptine / pharmacology
  • Corpus Striatum / metabolism*
  • Haloperidol / pharmacology
  • Humans
  • Male
  • Mice
  • Mice, Inbred C57BL
  • Receptors, Dopamine / drug effects
  • Receptors, Dopamine / metabolism*
  • Spiperone / metabolism
  • Stereotyped Behavior / drug effects
  • Time Factors

Substances

  • Receptors, Dopamine
  • Bromocriptine
  • Spiperone
  • Haloperidol
  • Apomorphine