Effects of prostaglandin E2 on the micronucleus formation in mouse bone marrow cells by various mutagens

Mutat Res. 1994 Dec 1;311(2):287-93. doi: 10.1016/0027-5107(94)90187-2.

Abstract

The effects of prostaglandin E2 (PGE2), as a trigger of erythroid progenitor cells into the cell cycle, were studied on the induction of micronuclei by various mutagens; with mitomicin C (MMC) the optimal protocol was established. PGE2 itself did not induce any micronuclei in this experiment. The highest frequency of micronuclei and dose-response relationship between PGE2 doses and micronucleus frequency were observed 30 h after injection of MMC to mice administered PGE2 24 h previously. Sensitization by PGE2 pretreatment was also found for other mutagens, such as vincristine, 5-fluorouracil, benzo[a]pyrene, 1,1-dimethylhydrazine and 2-naphthylamine. These results support the hypothesis that accelerating the erythropoiesis increases the frequency of micronuclei induced by mutagens.

MeSH terms

  • 2-Naphthylamine / toxicity
  • Animals
  • Benzo(a)pyrene / toxicity
  • Blood Cell Count
  • Bone Marrow / drug effects*
  • Bone Marrow / metabolism
  • Bone Marrow Cells
  • Dimethylhydrazines / toxicity
  • Dinoprostone / toxicity*
  • Dose-Response Relationship, Drug
  • Drug Synergism
  • Erythroblasts
  • Erythropoiesis / drug effects*
  • Erythropoietin / biosynthesis
  • Fluorouracil / toxicity
  • Male
  • Mice
  • Mice, Inbred BALB C
  • Micronucleus Tests*
  • Mitomycin / toxicity
  • Mutagens / toxicity*
  • Time Factors
  • Vincristine / toxicity

Substances

  • Dimethylhydrazines
  • Mutagens
  • Erythropoietin
  • Benzo(a)pyrene
  • dimazine
  • Mitomycin
  • Vincristine
  • 2-Naphthylamine
  • Dinoprostone
  • Fluorouracil