Interleukin-4 induces endothelial vascular cell adhesion molecule-1 (VCAM-1) by an NF-kappa b-independent mechanism

FEBS Lett. 1995 Sep 25;372(2-3):194-8. doi: 10.1016/0014-5793(95)00976-g.

Abstract

While all features of the inflammatory response induced by IL-1 are not observed following IL-4 stimulation, suboptimal concentrations both cytokines result in synergistic VCAM-1 expression in HUVEC. We have shown that, while IL-1 stimulated HUVEC express GM-CSF, tissue factor and VCAM-1, only VCAM-1 is detectable after exposure to IL-4. While kB was found essential for both basal and IL-1-mediated activity of VCAM-1, IL-4 induction was kB-independent. Inducible kB-binding proteins were identified in IL-1-, but not IL-4-stimulated nuclear extracts. Our results indicate that IL-4 exerts its transcriptional effects on the VCAM-1 gene through element(s) which do not require kB.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Cells, Cultured
  • Endothelium, Vascular / metabolism*
  • Humans
  • Interleukin-1 / pharmacology
  • Interleukin-4 / pharmacology*
  • NF-kappa B / metabolism
  • Transcription Factors / metabolism*
  • Vascular Cell Adhesion Molecule-1 / biosynthesis*

Substances

  • Interleukin-1
  • NF-kappa B
  • Transcription Factors
  • Vascular Cell Adhesion Molecule-1
  • Interleukin-4