Nitroglycerin-induced direct protection of the ischaemic myocardium in isolated working hearts of rats with vascular tolerance to nitroglycerin

Br J Pharmacol. 1995 Aug;115(7):1129-31. doi: 10.1111/j.1476-5381.1995.tb15014.x.

Abstract

We investigated whether nitroglycerin (NTG) was able to produce an anti-ischaemic effect in isolated working hearts of rats with vascular tolerance to NTG. Hearts isolated from tolerant and non-tolerant rats were subjected to 10 min coronary occlusion in the presence of 10(-7) M NTG and/or its solvent. NTG alleviated ischaemia-induced deterioration of cardiac function and decreased lactate dehydrogenase release whilst having no effect on coronary flow nor the area of the ischaemic zone both in hearts isolated from NTG-tolerant and non-tolerant rats. The magnitude of the effect was similar in the two groups. These results suggest that the anti-ischaemic effect of NTG involves direct myocardial mechanisms independent of its vascular action and that vascular tolerance to NTG does not affect this direct protective action.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Coronary Vessels / drug effects
  • Drug Tolerance
  • Heart / drug effects*
  • Heart / physiopathology
  • In Vitro Techniques
  • Male
  • Myocardial Ischemia / prevention & control*
  • Nitroglycerin / pharmacology*
  • Rats
  • Rats, Wistar
  • Vasodilator Agents / pharmacology*

Substances

  • Vasodilator Agents
  • Nitroglycerin