Abstract
An imbalance in TH1-type and TH2-type responses may allow Candida albicans to modify the host response to favor its own persistence. This hypothesis has important consequences for allergy, autoimmunity and co-infection, and also highlights a potential role for cytokine and anti-cytokine therapy in Candida-related pathology.
Publication types
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Research Support, Non-U.S. Gov't
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Review
MeSH terms
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Animals
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Candida albicans / immunology
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Candidiasis / drug therapy
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Candidiasis / immunology*
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Cytokines / antagonists & inhibitors
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Cytokines / immunology
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Cytokines / therapeutic use
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Humans
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Immune Tolerance
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Th1 Cells / immunology*
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Th2 Cells / immunology*