Signal transduction versus buffering activity in Ca(2+)-binding proteins

Nat Struct Biol. 1994 Apr;1(4):239-45. doi: 10.1038/nsb0494-239.

Abstract

The three-dimensional structure of calbindin D9k in the absence of Ca2+ has been determined using NMR spectroscopy in solution, allowing the first direct analysis of the consequences of Ca2+ binding for a member of the calmodulin superfamily of proteins. The overall response in calbindin D9k is much attenuated relative to the current model for calmodulin and troponin C. These results demonstrate a novel mechanism for modulating the conformational response to Ca(2+)-binding in calmodulin superfamily proteins and provide insights into how their Ca(2+)-binding domains can be fine-tuned to remain essentially intact or respond strongly to ion binding, in relation to their functional requirements.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Apoproteins / chemistry*
  • Apoproteins / metabolism
  • Binding Sites
  • Buffers
  • Calbindins
  • Calcium / metabolism
  • Cattle
  • Magnetic Resonance Spectroscopy
  • Models, Molecular
  • Molecular Structure
  • Protein Binding
  • Protein Conformation
  • Recombinant Proteins / chemistry
  • Recombinant Proteins / metabolism
  • S100 Calcium Binding Protein G / chemistry*
  • S100 Calcium Binding Protein G / metabolism
  • Signal Transduction*
  • Troponin / chemistry
  • Troponin C

Substances

  • Apoproteins
  • Buffers
  • Calbindins
  • Recombinant Proteins
  • S100 Calcium Binding Protein G
  • Troponin
  • Troponin C
  • Calcium