Phorbol esters induce nitric oxide synthase and increase arginine influx in cultured peritoneal macrophages

FEBS Lett. 1993 Apr 5;320(2):135-9. doi: 10.1016/0014-5793(93)80078-9.

Abstract

Incubation of peritoneal macrophages with beta-phorbol 12,13-dibutyrate promotes a time-dependent release of NO to the incubation medium. This effect was antagonized by LPS, a well known inducer of nitric oxide synthase (NOS) expression in macrophages, and was inhibited by NG-methyl-L-arginine and N omega-nitro-L-arginine. An increase in intracellular cGMP and NOS activity was observed in parallel with NO release. The induction of NOS was accompanied by a stimulation of arginine influx within the cell. These results suggest that activation of protein kinase C by phorbol esters is sufficient to promote NOS induction in macrophages.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amino Acid Oxidoreductases / biosynthesis*
  • Animals
  • Arginine / metabolism*
  • Cells, Cultured
  • Cyclic GMP / metabolism
  • Enzyme Activation
  • Enzyme Induction
  • Lipopolysaccharides / pharmacology
  • Macrophages / drug effects*
  • Macrophages / metabolism
  • Male
  • Nitric Oxide / metabolism*
  • Nitric Oxide Synthase
  • Peritoneal Cavity / cytology
  • Phorbol 12,13-Dibutyrate / pharmacology*
  • Protein Kinase C / metabolism
  • Rats

Substances

  • Lipopolysaccharides
  • Nitric Oxide
  • Phorbol 12,13-Dibutyrate
  • Arginine
  • Nitric Oxide Synthase
  • Amino Acid Oxidoreductases
  • Protein Kinase C
  • Cyclic GMP