On the role of the carboxyl group of sialic acid in binding of cholera toxin to the receptor glycosphingolipid, GM1

J Biochem. 1994 Dec;116(6):1269-74. doi: 10.1093/oxfordjournals.jbchem.a124674.

Abstract

The carboxyl group of the natural cholera toxin receptor, the ganglioside GM1, Gal beta 1-3GalNAc beta 1-4(NeuAc alpha 2-3)Gal beta 1-4Glc beta 1-Cer, has been converted to a number of C(1)-amides of NeuAc. The binding of cholera toxin B-subunit to these derivatives was monitored by exposing the modified glycolipids, on solid phases, to radiolabeled toxin. Binding was obtained, although substantially reduced, with the amide and to a lesser extent with the benzylamide and also the C(1)-alcohol. In the assay system used, the methyl-, ethyl-, or propylamides did not bind. It was concluded that the hydrogen bonding capacity of a carboxyl or amide group is needed for strong binding. This is in agreement with the recently published crystal structure of the B-subunit in complex with the GM1 pentasaccharide.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Binding Sites
  • Carbohydrate Sequence
  • Cholera Toxin / metabolism*
  • Chromatography, Ion Exchange
  • G(M1) Ganglioside / analogs & derivatives*
  • G(M1) Ganglioside / metabolism*
  • Glycosphingolipids / metabolism
  • Models, Molecular
  • Molecular Sequence Data
  • Peptide Fragments / metabolism
  • Protein Binding
  • Receptors, Cell Surface / metabolism*
  • Sialic Acids / metabolism*
  • Spectrometry, Mass, Fast Atom Bombardment

Substances

  • Glycosphingolipids
  • Peptide Fragments
  • Receptors, Cell Surface
  • Sialic Acids
  • choleragen receptor
  • glycolipid receptor
  • G(M1) Ganglioside
  • Cholera Toxin