Molecular pathogenesis of the chromosome 16 inversion in the M4Eo subtype of acute myeloid leukemia

Blood. 1995 May 1;85(9):2289-302.
No abstract available

Publication types

  • Comparative Study
  • Review

MeSH terms

  • Animals
  • Base Sequence
  • Biomarkers, Tumor / analysis
  • Cell Transformation, Neoplastic / genetics
  • Chromosome Fragility
  • Chromosome Inversion*
  • Chromosome Mapping
  • Chromosomes, Human, Pair 16 / ultrastructure*
  • Cloning, Molecular
  • Core Binding Factor Alpha 1 Subunit
  • DNA-Binding Proteins / genetics*
  • Eosinophils / pathology
  • Genes
  • Humans
  • Leukemia, Myelomonocytic, Acute / classification
  • Leukemia, Myelomonocytic, Acute / genetics*
  • Leukemia, Myelomonocytic, Acute / pathology
  • Mice
  • Models, Genetic
  • Molecular Sequence Data
  • Myosins / genetics*
  • Neoplasm Proteins*
  • RNA, Messenger / genetics
  • RNA, Neoplasm / genetics
  • Recombinant Fusion Proteins / genetics*
  • Species Specificity
  • Transcription Factor AP-2
  • Transcription Factors / genetics*

Substances

  • Biomarkers, Tumor
  • Core Binding Factor Alpha 1 Subunit
  • DNA-Binding Proteins
  • Neoplasm Proteins
  • RNA, Messenger
  • RNA, Neoplasm
  • RUNX2 protein, human
  • Recombinant Fusion Proteins
  • Runx2 protein, mouse
  • Transcription Factor AP-2
  • Transcription Factors
  • Myosins