Ca2+ release from mitochondria induces cytosolic enzyme leakage in anoxic liver

J Surg Res. 1995 May;58(5):485-90. doi: 10.1006/jsre.1995.1076.

Abstract

The mechanisms of elevation of cytosolic Ca2+ concentration during anoxia causing cytosolic enzyme leakage were studied in perfused rat liver. Before anoxia the Ca2+ contents in mitochondria of perfused rat liver were varied by addition of noradrenaline and glucagon, lowering Ca2+ concentration in the perfusion medium, and addition of Ca2+ channel blocker, either singly or in combination. The amount of cytosolic enzyme leakage during anoxic perfusion positively correlated with the mitochondrial Ca2+ content just before anoxia and also with Ca2+ release from mitochondria during anoxia. However, the amount of cytosolic enzyme leakage during anoxic perfusion did not correlate with the Ca2+ concentration in the perfusion medium and microsomal Ca2+ content. These data support the hypothesis that in anoxic liver, the cytosolic Ca2+ concentration is elevated to a pathological level mainly by Ca2+ release from mitochondria, causing the cytosolic enzyme leakage.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Calcium / metabolism*
  • Cytosol / enzymology*
  • Egtazic Acid / analogs & derivatives
  • Egtazic Acid / pharmacology
  • Hypoxia / metabolism*
  • Indicators and Reagents
  • Liver / metabolism*
  • Male
  • Microsomes, Liver / metabolism
  • Mitochondria, Liver / metabolism*
  • Perfusion
  • Phosphorylase a / metabolism
  • Rats
  • Rats, Sprague-Dawley

Substances

  • Indicators and Reagents
  • Egtazic Acid
  • Phosphorylase a
  • 1,2-bis(2-aminophenoxy)ethane-N,N,N',N'-tetraacetic acid
  • Calcium