Human PBMC proliferative response to Leishmania infantum promastigotes is inhibited by anti-IFN-gamma mAb gamma 123

New Microbiol. 1995 Jan;18(1):53-8.

Abstract

PBMC from individuals both exposed and non-exposed to leishmaniae proliferative and produce interferon-gamma (IFN-gamma) following stimulation with Leishmania antigens. We studied the kinetics of the proliferative response of PBMC from non-exposed individuals and from patients recovering from visceral leishmaniasis due to Leishmania infantum, using heat-killed stationary-phase promastigotes of L. infantum as stimulating agent. The kinetics of both groups followed a similar temporal pattern, with higher values in the patient's group. Moreover, we observed that in both groups the activation was dose-dependently inhibited following the addition of gamma 123 anti-IFN-gamma monoclonal antibody. These results indicate the need for IFN-gamma in the activation process of PBMC induced by Leishmania antigens and stress the role of IFN-gamma in the immune response to leishmaniasis. The relevance of the elucidation of the immune response mechanism in human leishmaniasis for therapy and vaccination is briefly discussed.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antibodies, Monoclonal / immunology
  • Antigens, Protozoan / immunology*
  • Humans
  • Interferon-gamma / biosynthesis
  • Interferon-gamma / immunology*
  • Interleukin-4 / biosynthesis
  • Interleukin-4 / immunology
  • Killer Cells, Natural / immunology
  • Kinetics
  • Leishmania infantum / immunology*
  • Leishmaniasis, Visceral / immunology*
  • Lymphocyte Activation*
  • T-Lymphocytes / immunology

Substances

  • Antibodies, Monoclonal
  • Antigens, Protozoan
  • Interleukin-4
  • Interferon-gamma