Transforming growth factor beta 1 induces apoptotic cell death in cultured human umbilical vein endothelial cells with down-regulated expression of bcl-2

Biochem Biophys Res Commun. 1995 May 25;210(3):1076-82. doi: 10.1006/bbrc.1995.1766.

Abstract

Transforming growth factor beta 1 (TGF-beta 1) is a potent growth inhibitor of human umbilical vein endothelial cells (HUVEC). We investigated whether apoptosis is involved in the TGF-beta 1 mediated HUVEC growth inhibition. Using immunofluorescence microscopy and staining by propidium iodide, we showed HUVEC treated with TGF-beta 1 contained characteristic apoptotic nuclei with condensation of chromatin. FACS analysis demonstrated that the number of cells with hypodiploid DNA contents were increased in HUVEC treated with TGF-beta 1. The DNA fragmentation assay exhibited typical ladder formation in TGF-beta 1-treated HUVEC. Using Western blot analysis, the expression of bcl-2 on HUVEC was down-regulated by TGF-beta 1-treatment. These findings suggest that TGF-beta 1 regulates the growth of HUVEC by inducing apoptosis accompanied with down-regulation of bcl-2 expression.

MeSH terms

  • Apoptosis / drug effects*
  • Cell Nucleus / drug effects
  • Cell Nucleus / ultrastructure
  • Cells, Cultured
  • DNA / drug effects
  • DNA / metabolism
  • DNA Damage
  • Dose-Response Relationship, Drug
  • Endothelium, Vascular / cytology
  • Endothelium, Vascular / drug effects*
  • Endothelium, Vascular / metabolism
  • Flow Cytometry
  • Gene Expression Regulation / drug effects*
  • Microscopy, Fluorescence
  • Protein-Tyrosine Kinases / biosynthesis
  • Proto-Oncogene Proteins / biosynthesis*
  • Proto-Oncogene Proteins c-bcl-2
  • Transforming Growth Factor beta / pharmacology*
  • Umbilical Veins

Substances

  • Proto-Oncogene Proteins
  • Proto-Oncogene Proteins c-bcl-2
  • Transforming Growth Factor beta
  • DNA
  • Protein-Tyrosine Kinases