Phospholipase C-mediated release of neuraminidase from Tritrichomonas foetus cell surface

Parasitol Res. 1995;81(3):188-92. doi: 10.1007/BF00937108.

Abstract

The release of the Tritrichomonas foetus plasma-membrane ectoenzyme neuraminidase by exogenous specific phospholipase C (PI-PLC) was investigated. Neuraminidase activity was determined using both the peanut agglutinin (PNA) hemagglutination test and the specific substrate N-acetylneuramin-lactose in a colorimetric assay. The release of the neuraminidase by PI-PLC was dependent on the reaction time and the concentration of PI-PLC. Neuraminidase activity was also detected in supernatant of untreated T. foetus. Spontaneous or PI-PLC-induced release of neuraminidase from protozoan cells was markedly decreased by 10 mM ZnCl2, suggesting the occurrence of an endogenous PI-PLC in the parasite. After T. foetus lysis at 37 degrees C with a solution of Triton X-114, neuraminidase activity was preferentially found in the aqueous phase rather than in the detergent phase, again suggesting that the parasite contains an endogenous PI-PLC that converts the hydrophobic form of neuraminidase anchored to the T. foetus cell membrane into a hydrophilic form. These results show that neuraminidase is linked to the T. foetus plasma membrane via a glycosylphosphatidylinositol anchor.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cell Membrane / enzymology
  • Chlorides / pharmacology
  • Glycosylphosphatidylinositols / metabolism
  • Kinetics
  • Neuraminidase / metabolism*
  • Tritrichomonas foetus / enzymology*
  • Type C Phospholipases / pharmacology*
  • Zinc Compounds / pharmacology

Substances

  • Chlorides
  • Glycosylphosphatidylinositols
  • Zinc Compounds
  • zinc chloride
  • Type C Phospholipases
  • Neuraminidase