Binding of Lambert-Eaton myasthenic syndrome IgG to synaptosomal proteins does not correlate with an inhibition of calcium uptake

Neurosci Lett. 1995 Mar 3;187(2):115-8. doi: 10.1016/0304-3940(95)11350-9.

Abstract

The immunochemical and functional properties of IgG fractions from patients with Lambert-Eaton myasthenic syndrome (LEMS) were examined in chick and rat synaptosomes. LEMS IgG immunoprecipitated 125I-omega conotoxin GVIA (125I-omega CgTx) labeled N-type calcium channels solubilized from both tissues, and reacted with a 65 kDa protein band in immunoblots of rat synaptosomes. Depolarization-induced 45Ca2+ influx into chick synaptosomes was partially inhibited by omega CgTx, whereas influx into rat synaptosomes was insensitive to omega CgTx. No effect of LEMS sera or IgG on 45Ca2+ uptake was apparent in either preparation.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Blotting, Western
  • Calcium / metabolism*
  • Calcium Channel Blockers / pharmacology
  • Calcium Channels / metabolism
  • Chickens
  • Humans
  • Immunoglobulin G / metabolism*
  • Lambert-Eaton Myasthenic Syndrome / blood*
  • Nerve Tissue Proteins / metabolism*
  • Peptides / pharmacology
  • Precipitin Tests
  • Rats
  • Synaptosomes / metabolism*
  • omega-Conotoxin GVIA

Substances

  • Calcium Channel Blockers
  • Calcium Channels
  • Immunoglobulin G
  • Nerve Tissue Proteins
  • Peptides
  • omega-Conotoxin GVIA
  • Calcium