High-dose peripheral inositol raises brain inositol levels and reverses behavioral effects of inositol depletion by lithium

Pharmacol Biochem Behav. 1994 Oct;49(2):341-3. doi: 10.1016/0091-3057(94)90431-6.

Abstract

Lithium (Li) reduces brain inositol levels. Berridge has suggested that this effect is related to Li's mechanism of action. It had previously been shown that pilocarpine causes a limbic seizure syndrome in lithium treated rats, and that these lithium-pilocarpine seizures are reversible by intracerebroventricular inositol administration to rats. We now show that although inositol passes the blood-brain barrier poorly, large doses of intraperitoneal (IP) inositol can also reverse Li-pilocarpine seizures. Using gas chromatography, IP inositol can raise brain inositol levels. Demonstration that inositol enters brain after peripheral administration provides a basis for possible pharmacological intervention in psychiatric disorders at the level of second messengers linked to the phosphatidylinositol cycle.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Behavior, Animal / drug effects*
  • Brain Chemistry / drug effects*
  • Cerebral Cortex / drug effects
  • Cerebral Cortex / metabolism
  • Chromatography, Gas
  • Glucose / metabolism
  • Injections, Intraperitoneal
  • Injections, Intraventricular
  • Inositol / antagonists & inhibitors
  • Inositol / pharmacokinetics*
  • Inositol / pharmacology
  • Lithium / antagonists & inhibitors*
  • Lithium / pharmacology
  • Male
  • Pilocarpine / antagonists & inhibitors
  • Pilocarpine / pharmacology
  • Rats
  • Rats, Sprague-Dawley
  • Seizures / chemically induced
  • Seizures / prevention & control

Substances

  • Pilocarpine
  • Inositol
  • Lithium
  • Glucose