Transfected human somatostatin receptor type 2, SSTR2, not only inhibits adenylate cyclase but also stimulates phospholipase C and Ca2+ mobilization

Biochem Biophys Res Commun. 1994 Apr 29;200(2):986-92. doi: 10.1006/bbrc.1994.1547.

Abstract

COS-7 cells were transfected with human somatostatin (SRIF) receptor type 1 and 2 (human SSTR1 and SSTR2, respectively) cDNAs. In human SSTR2-expressing cells, SRIF not only inhibited forskolin-induced cAMP accumulation but also stimulated phospholipase C and Ca2+ mobilization. While the inhibition of cAMP accumulation was completely reversed by pertussis toxin (PTX) treatment of the cells, SRIF-induced activation of phospholipase C and Ca2+ mobilization was partially but not completely inhibited by the toxin treatment. In human SSTR1-expressing cells, however, SRIF induced only slight inhibition of cAMP accumulation and stimulation of phospholipase C-Ca2+ system. We conclude that the transfected SSTR2 can couple to phospholipase C as well as adenylate cyclase in a stimulatory and inhibitory manner, respectively. Both PTX-sensitive and -insensitive GTP-binding proteins may be involved in the SSTR2 signal transduction mechanisms.

MeSH terms

  • Adenosine Triphosphate / pharmacology
  • Adenylate Cyclase Toxin
  • Adenylyl Cyclase Inhibitors*
  • Animals
  • Calcium / metabolism*
  • Cell Line
  • DNA, Complementary / genetics
  • GTP-Binding Proteins / metabolism
  • Humans
  • Pertussis Toxin
  • Receptors, Somatostatin / classification
  • Receptors, Somatostatin / genetics
  • Receptors, Somatostatin / metabolism*
  • Somatostatin / metabolism
  • Somatostatin / pharmacology
  • Transfection
  • Type C Phospholipases / metabolism*
  • Virulence Factors, Bordetella / pharmacology

Substances

  • Adenylate Cyclase Toxin
  • Adenylyl Cyclase Inhibitors
  • DNA, Complementary
  • Receptors, Somatostatin
  • Virulence Factors, Bordetella
  • Somatostatin
  • Adenosine Triphosphate
  • Pertussis Toxin
  • Type C Phospholipases
  • GTP-Binding Proteins
  • Calcium