CD4+ T cells require adhesion via LFA-1/ICAM-1 to induce target apoptosis in TNF-independent pathway

Cell Immunol. 1994 Jun;156(1):135-45. doi: 10.1006/cimm.1994.1159.

Abstract

An antigen-specific CD4+ T cell clone, OK2.21, and a hybridoma clone, A3.4C6, induced cytolysis and DNA fragmentation of appropriate targets in the presence of an anti-CD3 monoclonal antibody (Mab). In a double chamber system, OK2.21 killed TNF-sensitive targets that were separated from the T cells by a porous membrane. The killing was completely inhibited by anti-TNF antibody. On the other hand, neither OK2.21 nor A3.4C6 killed a TNF-insensitive target, A20.2J, that was separated from the T cells. Furthermore, the cytotoxicity and DNA fragmentation of A20.2J by anti-CD3 Mab-activated T cells were inhibited by both anti-LFA-1 and anti-ICAM-1 Mab. These results suggest that TNF is the only soluble cytolytic factor involved in CD4+ T cell-mediated cytotoxicity, and that TNF-independent apoptosis occurs through cell-to-cell contact via a LFA-1/ICAM-1 system.

MeSH terms

  • Animals
  • Apoptosis
  • CD3 Complex / immunology
  • CD4-Positive T-Lymphocytes / cytology*
  • Cell Adhesion
  • Cell Adhesion Molecules / physiology
  • Clone Cells
  • Cytotoxicity, Immunologic*
  • DNA Damage
  • Immunity, Cellular
  • Intercellular Adhesion Molecule-1
  • Lymphocyte Function-Associated Antigen-1 / physiology
  • Mice
  • Mice, Inbred BALB C
  • Tumor Necrosis Factor-alpha / pharmacology*

Substances

  • CD3 Complex
  • Cell Adhesion Molecules
  • Lymphocyte Function-Associated Antigen-1
  • Tumor Necrosis Factor-alpha
  • Intercellular Adhesion Molecule-1