Ectopic expression of the CCAAT/enhancer-binding protein alpha promotes the adipogenic program in a variety of mouse fibroblastic cells

Genes Dev. 1994 Jul 15;8(14):1654-63. doi: 10.1101/gad.8.14.1654.

Abstract

The CCAAT/enhancer-binding protein alpha (C/EBP alpha) has been implicated in the regulation of adipoblast differentiation. In this study we investigate the potential of C/EBP alpha to promote the adipogenic program in a variety of fibroblastic cells. Transduction of the C/EBP alpha gene into eight mouse fibroblastic cell lines by retroviruses and DNA transfection generates adipocyte colonies at variable frequencies. The most dramatic results are obtained with NIH-3T3 cells, in which the percentage of G418-resistant colonies that exhibit the adipocyte morphology is reproducibly > 50% when the C/EBP alpha gene is transduced by retroviruses. The ability to promote the adipogenic program requires the potent transcriptional activation domain of C/EBP alpha and is not observed with C/EBP beta. Paradoxically, in spite of its antimitogenic effects, clonal cell lines that stably express high amounts of C/EBP alpha can readily be generated. Stable expression of C/EBP alpha in BALB/c-3T3 cells dramatically enhances their ability to terminally differentiate into adipocytes. The results demonstrate that C/EBP alpha can efficiently promote the adipogenic program in a variety of mouse fibroblastic cells, including those that have little or no spontaneous capacity to undergo adipogenesis.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • 3T3 Cells
  • Adipocytes / cytology*
  • Animals
  • Base Sequence
  • CCAAT-Enhancer-Binding Proteins
  • Cell Differentiation*
  • Cell Line
  • Chloramphenicol O-Acetyltransferase / biosynthesis
  • DNA Primers
  • DNA-Binding Proteins / biosynthesis*
  • Fibroblasts / cytology
  • Fibroblasts / metabolism
  • Gene Expression*
  • Mice
  • Molecular Sequence Data
  • Nuclear Proteins / biosynthesis*
  • Polymerase Chain Reaction
  • Procollagen / biosynthesis
  • Promoter Regions, Genetic
  • RNA, Messenger / metabolism
  • Transfection

Substances

  • CCAAT-Enhancer-Binding Proteins
  • DNA Primers
  • DNA-Binding Proteins
  • Nuclear Proteins
  • Procollagen
  • RNA, Messenger
  • Chloramphenicol O-Acetyltransferase