Excision of cyclobutane dimers in genomic and episomal DNA in human cells

Photochem Photobiol. 1994 Sep;60(3):221-4. doi: 10.1111/j.1751-1097.1994.tb05094.x.

Abstract

Direct determination has been made of cyclobutyl pyrimidine dimer induction and excision repair in an episomal SV40 DNA population in vivo. Maintaining SV40-transformed human (GM637) cells in confluent culture results in amplification of a mutant SV40 episome to high copy number. T4 endonuclease V was used to quantify the induction and repair of cyclobutane dimers in the SV40 episome and genomic DNA of the same cells. Differences in both parameters were observed; cyclobutane dimers were induced at 1.5-2-fold greater frequency in episomal DNA and excised at a reduced rate compared to genomic DNA in the host cells.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Cell Line, Transformed
  • Chromosomes, Human / radiation effects*
  • DNA / isolation & purification
  • DNA / radiation effects*
  • DNA Repair*
  • DNA, Superhelical / radiation effects
  • DNA, Viral / isolation & purification
  • DNA, Viral / radiation effects*
  • Genome, Viral*
  • Humans
  • Pyrimidine Dimers*
  • Simian virus 40
  • Ultraviolet Rays*

Substances

  • DNA, Superhelical
  • DNA, Viral
  • Pyrimidine Dimers
  • DNA