ET-1 induced alterations of hepatic microcirculation: sinusoidal and extrasinusoidal sites of action

Am J Physiol. 1994 Jul;267(1 Pt 1):G143-9. doi: 10.1152/ajpgi.1994.267.1.G143.

Abstract

Using epifluorescence microscopy, we investigated the dynamic changes in hepatic sinusoidal hemodynamics in vivo during continuous infusion of endothelin-1 (ET-1) in pentobarbital-anesthetized rats. ET-1 was infused for 20 min at rates of 2 or 10 pmol/min either systemically or into the portal vein, followed by a 90-min recovery period. In contrast to systemic application of ET-1 that did not cause a consistent hepatic microvascular effect, we observed two different patterns of microcirculatory alterations during portal application. Infusion of 2 pmol/min elicited a rapid, reversible decrease in sinusoidal diameter that was paralleled by a slight increase in red cell velocity, resulting in conservation of volumetric flow and sinusoid density. Infusion of 10 pmol/min resulted in a biphasic narrowing followed by transient increase in sinusoidal diameter and a profound and lasting decrease in red cell velocity, leading to an almost complete cessation of hepatic microvascular blood flow. These results indicate that ET-1 is a potent constrictor in the liver microcirculation in vivo and acts at both extrasinusoidal and sinusoidal sites, although the sinusoidal sites appear to be more sensitive to lower concentrations.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Endothelins / pharmacology*
  • Hemodynamics / drug effects
  • Liver Circulation / drug effects*
  • Male
  • Microcirculation / drug effects
  • Microscopy, Fluorescence
  • Portal System / drug effects
  • Rats
  • Rats, Sprague-Dawley
  • Vasoconstriction

Substances

  • Endothelins