CD4+ lymphocyte depletion in HIV-infected patients is associated with gp120-immunoglobulin-complement attachment to CD4+ cells

Vox Sang. 1993;64(1):31-6. doi: 10.1111/j.1423-0410.1993.tb02511.x.

Abstract

The mechanism of CD4+ lymphocyte depletion, which is the main immunological feature in HIV-infected patients, is unclear. We investigated whether gp120-immunoglobulin-complement complexes on the surface of CD4+ cells might be involved in the elimination of CD4+ lymphocytes. The results obtained in 63 HIV-infected patients show that gp120 is attached to a variable degree to CD4+ cells. Importantly, the percentage of CD4+gp120+ lymphocytes is inversely associated with CD4+ lymphocyte counts in the peripheral blood (p = 0.0004). CD4+gp120+ blood lymphocytes bind IgM (p = 0.0027) and IgG antibodies (p = 0.0001) and complement (p = 0.0005). These results suggest that immune complex-mediated cell elimination is an important mechanism of CD4+ cell depletion in patients with AIDS.

MeSH terms

  • Antigen-Antibody Complex / immunology*
  • CD4-Positive T-Lymphocytes / cytology*
  • Complement System Proteins / immunology*
  • HIV Envelope Protein gp120 / immunology*
  • HIV Infections / immunology*
  • Humans
  • Immunoglobulins / immunology*
  • Leukocyte Count

Substances

  • Antigen-Antibody Complex
  • HIV Envelope Protein gp120
  • Immunoglobulins
  • Complement System Proteins